Inflammation, Immunity, and Hypertensive End-Organ Damage
نویسندگان
چکیده
منابع مشابه
Inflammation, immunity, and hypertensive end-organ damage.
For >50 years, it has been recognized that immunity contributes to hypertension. Recent data have defined an important role of T cells and various T cell-derived cytokines in several models of experimental hypertension. These studies have shown that stimuli like angiotensin II, deoxycorticosterone acetate-salt, and excessive catecholamines lead to formation of effector like T cells that infiltr...
متن کاملAldosterone and end-organ damage.
PURPOSE OF REVIEW This review highlights recent clinical studies demonstrating the contribution of aldosterone to cardiovascular mortality, vascular dysfunction, and renal injury in the context of advances in our understanding of the molecular biology of aldosterone. RECENT FINDINGS Mineralocorticoid receptor antagonism reduces mortality in patients with congestive heart failure and following...
متن کاملDoppler ultrasonography and hypertensive target organ damage.
Address for Correspondence: Akihiro Tojo, Division of Nephrology and Endocrinology, The University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, 113-8655 Tokyo-Japan Phone: 81-3-3815-5411 Ext. 37219 Fax: 81-3-3814-0021 E-mail: [email protected] Accepted Date: 15.11.2013 Available Online Date: 09.12.2013 ©Copyright 2014 by AVES Available online at www.anakarder.com doi:10.5152/akd.2013.12677 Editorial ...
متن کاملPulsed and Tissue Doppler Echocardiographic Changes in Hypertensive Crisis with and without End Organ Damage.
BACKGROUND Hypertensive crisis (HC) is a common medical emergency associated with acute rise in arterial blood pressure that leads to end-organ damage (EOD). Therefore, it is imperative to find markers that may help in the prediction of EOD in acute hypertensive crisis. AIM To assess the clinical presentations on admission; echocardiographic changes of pulsed and tissue Doppler changes in EOD...
متن کاملRelationship between baroreceptor reflex function and end-organ damage in spontaneously hypertensive rats.
The purpose of this study was to further illustrate the relationship between baroreceptor reflex sensitivity (BRS) and hypertensive end-organ damage (EOD) and to test the hypothesis that impairment of BRS aggravates EOD in hypertension. We studied baroreflex-mediated changes in heart rate [expressed as baroreceptor sensitivity to heart rate control (BRSHR)] and blood pressure [expressed as baro...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Circulation Research
سال: 2015
ISSN: 0009-7330,1524-4571
DOI: 10.1161/circresaha.116.303697